As the key DSB repair service pathway is mediated by HR (36–38), we examined development from the Rad51 and γ-H2AX foci and their colocalization in HT29 cells. As antibodies directed versus 53BP1 and Rad51 had been the two rabbit polyclonal antibodies and unsuitable for double staining, we used mouse anti–γ-H2AX https://johnl147yei4.wikipresses.com/user